Dynamin independent endoystosis is an alternative cell entry mechanism for multiple animal viruses.

Check out our latest publication!

“Dynamin independent endocytosis is an
alternative cell entry mechanism for multiple
animal viruses”

Don’t miss our latest study protocol by Ojha, Ravi et al. which has been published in the PLOS Pathogens Journal.

The study explores how certain viruses can enter host cells without relying on the protein dynamin, which is typically essential for endocytosis.

This study underscores the adaptability of viruses in hijacking host cellular machinery and suggests that targeting multiple entry mechanisms may be necessary for effective antiviral interventions.

For a more detailed exploration of the research, you can access the full article here: Long COVID publications.

Ojha, R., Jiang, A., Mäntylä, E., Quirin, T., Modhira, N., Witte, R., Gaudin, A., De Zanetti, L., Gormal, R. S., Vihinen-Ranta, M., Mercer, J., Suomalainen, M., Greber, U. F., Yamauchi, Y., Lozach, P. Y., Helenius, A., Vapalahti, O., Young, P., Watterson, D., Meunier, F. A., … Balistreri, G. (2024). Dynamin independent endocytosis is an alternative cell entry mechanism for multiple animal viruses. PLoS pathogens20(11), e1012690. https://doi.org/10.1371/journal.ppat.1012690